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home > slc25a39 is necessary for mitochondrial glutathione import in mammalian cells SLC25A39 binds and modulates PRDX1 to suppress ROS-induced necroptosis in hepatocellular carcinoma > slc25a39 is necessary for mitochondrial glutathione import in mammalian cells SLC25A39 binds and modulates PRDX1 to suppress ROS-induced necroptosis in hepatocellular carcinoma
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Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial glutathione homeostasis bioRxiv Autoregulatory control of mitochondrial glutathione homeostasis Science SLC25A39 identified as a key regulator of hepatocellular carcinoma progression through the mitochondrial ROScytochrome ccaspase signaling axis Cellular & Molecular Biology Letters Springer Nature Link Ironing out the details: ferroptosis and its relevance to diabetic cardiomyopathy American Journal of Physiology Regulatory, Integrative and Comparative Physiology American Physiological Society
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Dual regulation of SLC25A39 by AFG3L2 and iron controls mitochondrial  glutathione homeostasis | bioRxiv
Autoregulatory control of mitochondrial glutathione homeostasis | Science
SLC25A39 identified as a key regulator of hepatocellular carcinoma  progression through the mitochondrial ROScytochrome ccaspase signaling  axis | Cellular & Molecular Biology Letters | Springer Nature Link
Ironing out the details: ferroptosis and its relevance to diabetic  cardiomyopathy | American Journal of Physiology-Regulatory, Integrative and  Comparative Physiology | American Physiological Society

slc25a39 is necessary for mitochondrial glutathione import in mammalian cells SLC25A39 binds and modulates PRDX1 to suppress ROS-induced necroptosis in hepatocellular carcinoma

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